MLXIPL promotes the migration, invasion, and glycolysis of hepatocellular carcinoma cells by phosphorylation of mTOR
Abstract Background Hepatocellular carcinoma (HCC) is associated with a high occurrence, mortality, and poor prognosis.MLX interacting protein like (MLXIPL) is an important regulator of glucolipid metabolism and is involved in tumor operation igloo white progression.We aimed to clarify the role of MLXIPL in HCC and its underlying mechanisms.Methods